Our non-drug alternative approach to the treatment of
vertiginous conditions is based on current accepted
understandings of the central control mechanisms involved in
movement, balance, and posture.
The sensory mismatch associated with dizziness or
spinning, may require stimulation and rehabilitation of
sensory systems other than the vestibular system. This
observation may explain why standard approaches to
vestibular rehabilitation sometimes fail. We are successful
in managing many types of vertigo, from BPPV to Chronic
Vestibulopathy.
Some types of vertigo may require only one visit. For a
consultation contact me toll free at 1-877-Brain-90 or
1-877-272-4690
Diagnostic procedures include a thorough Neurological
evaluation including Video Electronystagmography.
 
Treatment
Treatment for vertigo
depends on identifying and eliminating the underlying cause.
If a particular medication is responsible for the condition,
lowering the dosage or discontinuing the drug may eliminate
vertigo.
Vestibular
Rehabilitation Therapy
Vestibular rehabilitation therapy (VRT) is a type of
physical therapy used to treat vertigo. The goal of
treatment is to minimize vertigo, improve balance, and
prevent falls by restoring normal function of the vestibular
system.
In VRT, the patient
performs exercises designed to allow the brain to adapt to
and compensate for whatever is causing the vertigo. The
success of this treatment depends on several factors
including the following:
- Age
of the patient
-
Cognitive function (e.g., memory, ability to follow
directions in order)
-
Coordination and motor skills
-
Overall health of the patient (including the central
nervous system)
-
Physical strength
Vestibular rehabilitation
therapy is designed by a physical therapist under the
direction of a physician. In most cases, patients visit the
therapist on a limited basis and perform custom-designed
exercises at home, several times a day. As the patient
progresses, difficulty of the exercises increases until the
highest level of balance is attained during head movement,
eye movement (i.e., tracking with the eyes), and walking.
Causes
Vertigo usually results
from a disorder in the peripheral vestibular system
(i.e., structures of the inner ear). It also may occur as a
result of a disorder in the central vestibular system
(i.e., vestibular nerve, brainstem, and cerebellum). In some
cases, the cause of vertigo is unknown.
Peripheral vestibular
disorders include
the following:
-
Benign paroxysmal positional vertigo (BPPV; most common
peripheral disorder; may be accompanied by hearing loss,
reduced cognitive function, and facial muscle weakness)
-
Ménière disease (fluctuating pressure of inner ear fluid
[endolymph]; results in severe vertigo, ringing in the
ears [tinnitus], and progressive hearing loss)
-
Ototoxicity (i.e., ear poisoning)
-
Vestibular neuritis (inflammation of vestibular nerve
cells; may be caused by viral infection)
Benign paroxysmal positional
vertigo occurs when
debris made up of calcium carbonate and protein (called
otoliths or ear crystals) builds up in and damages the inner
ear. Inner ear degeneration (usually occurs in elderly
patients), head trauma, and inner ear infection
(e.g., otitis media, labyrinthitis) can cause BPPV.
Some medications and
environmental chemicals (e.g., lead, mercury, tin)
can cause ototoxicity (i.e., ear poisoning), which may
result in damage to the inner ear or the 8th cranial nerve
(acoustic nerve) and cause vertigo. The damage can be
permanent or temporary.
Long-term use or high doses
of certain antibiotics (e.g., aminoglycosides
[streptomycin, gentamicin]) and antineoplastics
(e.g., cisplatin, carboplatin) can cause permanent
ototoxicity.
Medications that may cause
temporary ototoxicity include the following:
-
Anticonvulsants (e.g., phenytoin, carbamazepine)
-
Antidepressants (e.g., clomipramine, amoxapine)
-
Antihypertensives (e.g., labetalol, enalapril)
- Loop
diuretics (e.g., bumetanide, furosemide)
- Pain
relievers (e.g., aspirin)
-
Prescription and over-the-counter cold medicines
-
Quinine (e.g., chloroquine, quinidine)
Alcohol, even in
small amounts, can cause temporary vertigo in some people.
Central vestibular
disorders that may
cause vertigo include the following:
-
Cardiovascular disorders (e.g., bradycardia [slowed
heart rate], tachycardia [rapid heart rate])
-
Central nervous system (CNS) disorders (e.g., stroke,
brain hemorrhage)
- Head
trauma
-
Migraine (30–50% of patients experience vertigo)
-
Multiple sclerosis (MS; may occur when demyelination
affects the brainstem or cerebellum)
-
Orthostatic hypotension (sharp decrease in blood
pressure upon rising from a lying or sitting position to
a standing position; caused by diabetes, dehydration,
and anemia)
-
Systemic diseases (e.g., kidney disease, thyroid
disorders)
-
Tumors that affect the central vestibular system (e.g.,
acoustic neuroma)
Overview
Diagnosis
It is important to diagnose
the cause of vertigo as quickly as possible to rule out
serious conditions such as cardiovascular disease, stroke,
hemorrhage, or tumor. Diagnosis includes clinical history,
physical and neurological examination, blood tests, and
imaging tests (e.g., CT scan, MRI scan).
Important considerations
include the following:
- What
triggers the vertigo?
- What
other symptoms occur?
- How
long do they last?
- What
improves or worsens symptoms?
Physical
examination includes measuring blood pressure and
heart rate . Neurological examination includes testing
facial and vestibular nerves and muscles, strength,
coordination, balance, and walking (gait).
The positional vertigo
test is used to help distinguish peripheral from central
vestibular disorders. In this test, the patient sits on a
table with the head turned to the side. The physician then
supports the head and lowers it gently below the table while
the patient lies back. The patient reports symptoms of
vertigo while the physician looks for circular movement of
the eyes (called nystagmus).
A delay between the onset
of nystagmus and the sensation of vertigo usually indicates
a peripheral vestibular disorder. Lack of a delay may
indicate a central vestibular disorder. The test is repeated
with the head turned in the opposite direction.
Electronystagmography
(ENG) is a neurological test used to evaluate the vestibular
system. It involves testing hearing in both ears (audiometry
tests), testing eye movements, and evaluating responses to
changes in posture and position.
Blood tests
include a complete blood count (CBC) and kidney and thyroid
panels to rule out systemic diseases (e.g., kidney disease,
hypothyroidism). If the patient is taking medication, drug
levels are obtained.
Imaging tests
may be used to detect brain abnormalities (e.g., stroke,
tumor). Computed tomography (CT scan) produces x-ray images
of the brain and magnetic resonance imaging (MRI) uses a
magnetic field to produce detailed images of brain tissue
and arteries in the neck and brain.
Signs
and Symptoms
Vertigo refers to the
sensation of spinning (subjective vertigo) or the
perception that surrounding objects are moving or spinning
(objective vertigo). Some patients describe a feeling of
being pulled toward the floor or toward one side of the
room. Moving the head, changing position, and turning while
lying down often worsen vertigo.
The sudden onset of vertigo
usually indicates a peripheral vestibular disorder (e.g.,
BPPV, Ménière disease, vestibular neuritis).
Symptoms of benign
paroxysmal positional vertigo (BPPV) usually last a few
seconds to a few minutes and are intermittent (i.e., come
and go). They also may include lightheadedness, imbalance,
and nausea, usually as a result of a change in position
(e.g., rolling over in bed, getting out of bed).
Symptoms of Ménière disease
and vestibular neuritis include vertigo, hearing loss,
ringing in the ears (tinnitus), and ear pressure that often
lasts hours to days.
Peripheral vestibular
disorders also may cause the following symptoms:
-
Blurred vision
-
Fatigue and reduced stamina
-
Headache
-
Heart palpitations (rapid fluttering of the heart)
-
Imbalance
-
Inability to concentrate
-
Increased risk for motion sickness
-
Muscle ache (especially of the neck and back)
-
Nausea and vomiting
-
Reduced cognitive function (i.e., thinking and memory)
-
Sensitivity to bright lights and noise
-
Sweating
Vertigo
caused by a central vestibular disorder usually
develops gradually. In addition to vertigo, central
vestibular disorders (e.g., stroke, migraine) may cause the
following symptoms:
-
Double vision (diplopia)
-
Headache (may be severe)
-
Impaired consciousness
-
Inability to speak due to muscle impairment (dysarthria)
- Lack
of coordination
-
Nausea and vomiting
-
Weakness
Complications
Severe vertigo can be
disabling and may result in complications such as
irritability, loss of self-esteem, depression, and injuries
from falls. Falls are the leading cause of serious injury in
people over the age of 65.
What is a Neurological
Exam?
The human
nervous system is an intricate and complex network of fibers
that impenetrates the entire body and functions in
complicated and often mysterious ways. Sophisticated imaging
and laboratory tests do not always provide sufficient
information about how the nerves are functioning -- or not
functioning, as the case may be. The neurological
examination is a series of simple questions and tests that
provide crucial information about the nervous system. It is
an inexpensive, noninvasive way to determine what might be
wrong.
The neurological
examination is divided into several components, each
focusing on a different part of the nervous system:
-
mental status
-
cranial nerves
-
motor system
-
sensory system
- the
deep tendon reflexes
-
coordination and the cerebellum
- gait
The exam
requires skill, patience, and intelligence on the part of
the physician, and cooperation from the patient. Incomplete
or inaccurate exams can lead to incorrect diagnoses.
Mental
status
The mental status examination is a series of detailed but
simple questions designed to test cognitive ability,
including the patient's:
-
state of consciousness (awareness and responsiveness to
the environment and the senses);
-
appearance and general behavior;
-
mood;
-
content of thought; and
-
intellectual resources (orientation with reference to
time, place, and person; comprehension; ability to pay
attention; insight; memory; judgment; abstract reasoning
power; speech and language function; and intellectual
capacity).
The
patient may be asked to remember objects that had been
listed earlier in the course of the exam; repeat sentences;
solve simple mathematical problems; copy a three-dimensional
drawing; and draw a clock and place the numbers and hands
appropriately. When speech and language are tested, the
examiner listens to the character of the speech, the fluency
(smoothness of speech), and the patient’s ability to
understand and carry out simple or complex commands, and to
read and write.
In addition to specific
questions that make up the actual mental status exam, the
neurologist obtains important information by observing the
patient’s general behavior during the examination.
Many neurological diseases,
such as dementia, cause changes in intellectual status or
emotional responsiveness, and specific personality features.
These changes and features can be detected during the mental
status portion of the neurological exam.
The mental status exam is
especially important when the other parts of the
neurological exam reveal no abnormalities. Sometimes, slight
changes in memory or other intellectual resources may be the
only indication that something is wrong. Evaluating a
person’s intellectual capacity can also be helpful in
determining a course of treatment and making a prognosis.
Cranial nerves
The cranial nerves are a set of 12 nerves that relay
messages between the brain and the head and neck and control
motor and sensory functions, including vision, smell, and
movement of the tongue and vocal cords.
The cranial nerve exam
involves testing the function of all 12 sets of cranial
nerves. It is an essential part of the neurological exam,
and helps localize central nervous system dysfunction and
aids in diagnosing systemic disease. Some of the functions
that are commonly tested as part of the cranial nerve exam
include: eyelid strength and function; visual function;
peripheral vision; pupillary light reflexes; eye muscle
movements; strength of facial musculature; the gag reflex;
tongue and lip movements; ability to smell and taste;
hearing; and sensation in the face, head, and neck.
Motor
system
The motor system includes the brain and spinal cord motor
pathways, and all the motor nerves and muscles throughout
the body. Abnormalities in the motor system can often be
detected by assessing muscle strength and tone and by
looking for a variety of characteristic signs.
The patient is usually
asked to undress, so the neurologist can see the muscles and
look for atrophy (shrinkage), twitching, or abnormal
movements. Tests are done to evaluate strength in all the
major muscle groups.
Evaluating Babinski
response is an important part of testing the motor system.
The neurologist strokes or scratches, heel-to-toe, the outer
side of the sole of the foot and in patients over the age of
2, the toes normally curl downward in response. If the toes
fan upward, a brain or spinal cord injury is indicated. A
number of neurological disorders can lead to Babinski
response.
Sensory system
Sensation depends on impulses that occur as a result of
stimulation of receptors located in the skin, muscles,
tendons, and so on, and are sent along nerve fibers to the
central nervous system (brain and spinal cord). The sensory
exam is used to determine areas of abnormal sensation, the
quality and type of sensation impairment, and the degree and
extent of tissue involvement.
A sensory exam involves
evaluating different types of sensation, including pain,
temperature, pressure, and position. For example, pinpricks
may be used to test the patient's response to pain and
compare the response in different parts or opposite sides of
the body. A cold or warm object may be used to test the
sensation of temperature. To test position, patients may be
asked to close their eyes and determine in which direction
the examiner is moving a part of their body (e.g., big toe).
Patients also may be asked to identify objects with their
eyes closed or identify numbers or letters traced on their
body.
The sensory exam should be
repeated to provide accurate results. Responses may be
affected by how alert, aware, and well-rested the patient
is, so this part of the neurological exam is usually
performed early in the course of testing.
Deep
tendon reflexes
Reflexes are actions performed involuntarily in response to
impulses sent to the central nervous system. Alterations in
reflexes are often the first sign of neurological
dysfunction. Observing reflexes is the most objective part
of the neurological exam, since the reflexes are not under
voluntary control and testing does not depend on the
patient’s cooperation, attitude, or awareness.
Hundreds of reflexes have
been identified, but the neurological exam generally
involves testing only the deep tendon reflexes. Deep tendon
reflexes, also known as muscle stretch reflexes, are
reflexes elicited in response to stimuli to tendons.
Normally, when a specific area of the muscle tendon is
tapped with a soft rubber hammer, the muscle fibers
contract. Abnormal responses may indicate injury to the
nervous system pathways that produce the deep tendon reflex.
Coordination and the cerebellum
The cerebellum is the part of the brain that controls
voluntary movement and motor coordination, including
posture. Testing coordination provides clues about
conditions that affect the cerebellum.
The neurologist may ask
patients to move their finger from their nose to the
neurologist’s finger, going back and forth from nose to
finger, touching the tip of each. Patients also may be asked
to tap their fingers together quickly in a coordinated
fashion or move their hands one on top of the other, back
and forth, as smoothly as they can. Coordination in the
lower limbs can be tested by asking patients to rub one heel
up and down smoothly over the other shin.
Gait
Most of us take our ability to walk for granted. But as
simple as it may seem, walking is a very intricate
physiological process. How we walk – our gait - is
influenced by a number of bodily mechanisms and nervous
system reflexes. The body must be held erect; the limbs,
head, and trunk must be held in the right position; the
person must be oriented to the position of all body parts;
parts of motor control involved with moving must be
integrated; and so on. Because walking depends on so many
different parts of the nervous system, it can be affected by
a variety of neurological disorders.
By observing gait, the
neurologist can gather important clues about what might be
wrong. The patient is usually asked to walk in different
ways (e.g., heel-to-toe in a straight line, turning
abruptly, walking on the toes, walking on the heels,
running).
References
Haerer, A.F., 1992, The Neurological Exam, J.B.
Lippincott Co.,(Courtesy of the Neurology Channel)
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